HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS		QUICK SEARCH:   [advanced] Author: Keyword(s): Year:  Vol:  Page:

This Article Figures Only Full Text Full Text (PDF) CME Test (opens in a new window) Submit a response Alert me when this article is cited Alert me when eLetters are posted Alert me if a correction is posted Citation Map Services Email this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal Download to citation manager Citing Articles Citing Articles via HighWire Citing Articles via Google Scholar Google Scholar Articles by Yoshimitsu, K. Articles by Masuda, K. Search for Related Content PubMed PubMed Citation Articles by Yoshimitsu, K. Articles by Masuda, K. Related Collections Computed Tomography Gastrointestinal Radiology

Unusual Hemodynamics and Pseudolesions of the Noncirrhotic Liver at CT¹

¹ From the Department of Clinical Radiology, Graduate School of Medical Sciences, Kyushu University, 3-1-1 Maisdashi, Higashi-ku, Fukuoka 812-8582, Japan. Presented as an education exhibit at the 2000 RSNA scientific assembly. Received February 2, 2001; revision requested March 9 and received April 16; accepted April 25. Address correspondence to K.Y. (e-mail: yoshimitsu@dr.hosp.kyushu-u.ac.jp).

Recognition of pseudolesions of the liver at computed tomography (CT) is important because of their close resemblance to primary liver cancers or metastases. Two types of pseudolesion in the noncirrhotic liver include that due to transient extrinsic compression, typically caused by ribs or the diaphragm, and that due to a "third inflow" of blood from other than the usual hepatic arterial and portal venous sources: the cholecystic, parabiliary, or epigastric-paraumbilical venous system. Although the location of both types of pseudolesion are characteristic, their appearances at CT during arterial portography and CT during selective angiography vary from nonenhanced low-attenuation areas to well-enhanced high-attenuation areas, depending on the amount and timing of the inflow and presence or absence of focal metabolic alteration of the hepatocytes. Radiologists need to understand the underlying mechanism of these pseudolesions to better recognize the wide range of their appearances at CT.

Index Terms: Computed tomography (CT), perfusion study, 761.12114 • Liver, abnormalities, 761.91 • Liver, blood supply, 958.12914 • Liver, CT, 761.12114

This article has been cited by other articles:

				J. W. Lee, S. Kim, S. W. Kwack, C. W. Kim, T. Y. Moon, S. H. Lee, M. Cho, D. H. Kang, and G. H. Kim Hepatic Capsular and Subcapsular Pathologic Conditions: Demonstration with CT and MR Imaging RadioGraphics, September 1, 2008; 28(5): 1307 - 1323. [Abstract] [Full Text] [PDF]

				L.-P. Liu, B.-W. Dong, X.-L. Yu, D.-K. Zhang, X. Li, and H. Li Analysis of Focal Spared Areas in Fatty Liver Using Color Doppler Imaging and Contrast-Enhanced Microvessel Display Sonography J. Ultrasound Med., March 1, 2008; 27(3): 387 - 394. [Abstract] [Full Text] [PDF]

				M. Macari, R. Yeretsian, and J. Babb Assessment of Low Signal Adjacent to the Falciform Ligament on Contrast-Enhanced MRI Am. J. Roentgenol., December 1, 2007; 189(6): 1443 - 1448. [Abstract] [Full Text] [PDF]

				S. Colagrande, N. Centi, R. Galdiero, and A. Ragozzino Transient Hepatic Intensity Differences: Part 2, Those Not Associated with Focal Lesions Am. J. Roentgenol., January 1, 2007; 188(1): 160 - 166. [Abstract] [Full Text] [PDF]

				K. Khalili, F. P. Lan, A. E. Hanbidge, D. Muradali, D. G. Oreopoulos, and I. R. Wanless Hepatic Subcapsular Steatosis in Response to Intraperitoneal Insulin Delivery: CT Findings and Prevalence Am. J. Roentgenol., June 1, 2003; 180(6): 1601 - 1604. [Abstract] [Full Text] [PDF]

				G. C. Mueller, H. K. Hussain, R. C. Carlos, H. V. Nghiem, and I. R. Francis Effectiveness of MR Imaging in Characterizing Small Hepatic Lesions: Routine Versus Expert Interpretation Am. J. Roentgenol., March 1, 2003; 180(3): 673 - 680. [Abstract] [Full Text] [PDF]